Attention-deficit hyperactivity disorder (ADHD) is a common chronic condition  that affects millions of children all over the world and often continues into adulthood. ADHD includes a combination of persistent problems, such as difficulty sustaining attention, hyperactivity and impulsive behaviour,Angerness  it also affects a person’s ability to concentrate and makes them easily distracted, impulsive, or excessively energetic. There are several medical  treatments available for ADHD, but another approach is to try to control symptoms through home means, such as your diet.  A Health, food, and nutrition can make a remarkable difference in the lives of both children and adults who have been diagnosed with ADHD

Some research has indicated that genetic factors, family education, environment and dietary habits have some major association with people suffering from ADHD. If You have attention deficit hyperactivity disorder then , you may notice that you have strong and very unpredictable  emotional reactions to things that other people seem to take in stride. Heightened, over-the-top emotions with the disorder are very common for people carrying this disease , and the reaction can be in both positive and negative situations. It is also not unusual for person  with ADHD to feel physically hypersensitive to touch, sounds, light, even of the tags on clothing. 

Studies have consistently documented that improvement with age in core symptoms are mostly accompanied by functional changes in cortical and cerebellar regions of the brain whereas basal ganglia anomalies seem to be a peculiar lifetime feature of ADHD, regardless of adult remission . Studies on structural anomalies and age-by-diagnosis interaction in the caudate and putamen have also supported the pivotal role of subcortical basal ganglia anomalies in the pathophysiology of ADHD which in turn gives observation about infections response of the patients . 

Probably, a variable and complex interaction between predisposing genetic and environmental factors during the early months or years of life could affect the normal path of neurodevelopment of a child . Considering the above-mentioned structural and functional evidences, environmental factors influencing basal ganglia development should be worthy of investigation and further studies . For instance, a specific vascular vulnerability of the striatum due to its positioning in a border zone of arterial supply has already been suggested by a case-control study reporting on hypoxic and other perinatal complications  and by the review of some in vivo studies . A more recent study has shown  the negative impact of exposure to polycyclic aromatic hydrocarbons on the caudate nucleus in people suffering from ADHD. 

Recently It has been reported that in China many children with ADHD also have allergic rhinitis or asthma. These children are more Venerable and   susceptible to the common cold or upper respiratory infections compared with normal healthy children. Additionally, the common cold or an upper respiratory infection may lead to some disease recurrence or worsen the symptoms of disorder in these children. Previous studies and some deep researches have determined that the ADHD may have a close association with allergic disease. Based on the clinically observed phenomenon and  studies, it was hypothesized that ADHD is a high inflammation  and immune‑associated disease. 

Therefore, some scientists designed a clinical and animal experiments to test this hypothesis in the future. Immune system disorders may be a intruding  part of the entomology  of ADHD. The current report and outcomes of experiments may have implications for future clinical practices. 

So the Patients with ADHD might be more prone to infections during there early childhood  stage and subclinical streptococcal infections during adulthood. Moreover, they seem to have an increased risk for basal ganglia autoimmunity by reaching adulthood. Both of these infections and the ensuing acquired autoimmunity could influence the neurodevelopmental process, by contributing, at least in part, to the ADHD pathogenesis.